Frank W. Rozsa, Ph.D.
Publications

Rozsa FW, Scott K, Pawar H, Moroi S, Richards JE. Effects of Timolol on MYOC, OPTN, and WDR36 RNA Levels. Arch Ophthalmol 2008;126:86-93.

Rozsa FW, Scott KM, Pawar H, Samples JR, Wirtz MK, Richards JE. Differential expression profile prioritization of positional candidate glaucoma genes: the GLC1C locus. Arch Ophthalmol 2007;125:117-127.

Rozsa FW, Reed DM, Scott KM, Pawar H, Moroi SE, Kijek TG, Krafchak CM, Othman MI, Vollrath D, Elner VM, Richards JE. Gene expression profile of human trabecular meshwork cells in response to long-term dexamethasone exposure. Mol Vis 2006;12:125-141.

Krafchak CM, Pawar H, Moroi SE, et al. Mutations in TCF8 cause posterior polymorphous corneal dystrophy and ectopic expression of COL4A3 by corneal endothelial cells. Am J Hum Genet 2005;77:694-708.

Shimizu S, Lichter, PR, Johnson PT, et al. Age-dependent prevalence of mutations at the GLC1A Locus in Primary Open-angle Glaucoma. Am J Ophthalmol 2000;130:165-177.

Zimmerman CC, Lingappa VR, Richards JE, et al. A trabecular meshwork glucocorticoid response (TIGR) gene mutation affects translocational processing. Mol Vis 1999;5:19. Available from: http://www.molvis.org/molvis/v5/p19

Richards JE, Ritch R, Lichter PR, et al. Novel trabecular meshwork inducible glucocorticoid response mutation in an eight-generation juvenile-onset primary open-angle glaucoma pedigree. Ophthalmology 1998;105:1698-1707.

Rozsa FW, Shimizu S, Lichter PR, et al. GLC1A mutations point to regions of potential functional importance on the TIGR/MYOC protein. Mol Vis 1998;4:20. Available from: http://www.molvis.org/molvis/v4/p20